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Moreover, given that we detected an increases in the levels of FXR-antagonistic BAs, but did not observe an elevation in the levels of TGR5-agonistic BAs and cAMP, we then speculate that FXR-mediated pathway rather than TGR5-mediated pathway was critically involved in the regulation of GLP-1 by T3 treatment in mice
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Either peripheral administration of leptin [349] or gastric balloon distention [350] acutely activates GLP-1-producing neurons in the NTS, as assessed by cFos immunoreactivity
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